PROPIEDADES FISIOLÓGICAS DEL MÚSCULO CARDIACO

PRACTICA DE LABORATORIO Nº 06 “PROPIEDADES FISIOLÓGICAS DEL MÚSCULO CARDIACO”

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SECUENCIA DE LA ACTIVIDAD DE LAS DIFERENTES PARTES DEL CORAZÓN

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SISTEMA DE CONDUCCIÓN O NODAL

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ntonces ¿ mo tra a a e corazón? 

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Control Nervioso

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Se hizo un registro basal de más o menos 20 latidos cardiacos y manteniendo el kimógrafo a la velocidad de 768mm por minuto. Estimulación del nervio vago derecho, durante 5 segundos con un estímulo de 5 a 10 voltios y una frecuencia de 10/seg.

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Las fibras parasimpáticas inervan los tejidos auriculares y de conducción..

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La inervación autónoma del corazón presenta una situación bilateral, ya que los nervios

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• La estimulación nerviosa hace que que se libere la hormona Ach en las terminaciones nerviosas, la cual produce dos

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Acción de La Acetilcolina

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Respuesta al impulso colinérgico

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FACTORES DE LA EXCITABILIDAD MIOCÁRDICA FRÍO

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Propagación de la excitación cardiaca • Las fibras miocárdicas tienen un potencial de membrana en reposo cercano a -90mV. Éstas fibras individuales están separadas por  membranas, pero la despolarización se propaga en forma radial a través de ellas como si fueran un sincicio por la presencia de nexos. . El potencial de acción transmembrana de las células musculares cardiacas aisladas se caracteriza por despolarización rápida, una meseta y un proceso lento de repolarización. • La despolarización inicial se debe a la entrada de Na+ a través de canales del Na+ de abertura

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Propagación de la excitación cardiaca •

La despolarización iniciada en el nodo SA se propaga en sentido radial por las aurículas, luego converge en el nodo AV. La despolarización auricular se completa en 0.1 s. Debido a que la conducción del nodo AV es lenta, hay un retraso aproximado de 0.1 s (retardo nodal AV) antes de que la excitación se propague a los ventrículos. Este retraso se acorta con la estimulación de los nervios simpáticos del corazón y se prolonga con la estimulación de los nervios vagos. Además, la despolarización del músculo ventricular  comienza en el lado izquierdo del tabique interventricular y se dirige primero a la derecha a través de la parte media del tabique. La ola de despolarización se extiende luego por el tabique hasta la punta

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• El efecto de la dismunición de la temperatura (frío) va producir una gran disminución de la frecuencia cardiaca, que puede disminuir  hasta sólo algunos latidos por minuto cuando una persona está cerca de la muerte por hipotermia en el intervalo de temperatura corporal de 16ºC a 21ºC. Estos efectos probablemente se deben al hecho de que el frío disminuye la permeabilidad de la membrana del músculo cardiaco a los iones que controlan la frecuencia cardiaca, disminuyendo el proceso de autoexcitación; es decir  cuando disminuye la frecuencia cardiaca, la relación corriente de Ca2+/tiempo es menor (menos PA), lo que deja mucho tiempo menos PA para que el Ca2+ salga. De este modo, se consigue una menor concentración citosólica media de Ca2+ y se mantiene una

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CALOR • El aumento de la temperatura corporal, como ocurre durante la fiebre, produce un gran aumento de la frecuencia cardiaca, a veces hasta el doble del valor  normal. • Estos efectos probablemente se deben al hecho de que el calor aumenta la permeabilidad de la membrana del músculo cardiaco a los iones que controlan la frecuencia cardiaca, acelerando el proceso de autoexcitación. • La fuerza contráctil del corazón con frecuencia aumenta

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EFECTO DEL ION POTASIO EN EL MIOCARDIO

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Estos efectos se deben parcialmente al hecho de que una concentración

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• b) ACCION DE LOS IONES CALCIO Un exceso de los iones calcio produce efectos casi exactamente contrarios a los iones potasio, haciendo que el corazón progrese hacia una contracción espastica. Esto esta producido por el efecto de los iones calcio en el inicio del proceso contráctil cardiaco.