Hemodynamic Disorders, Thromboembolic Disease and Shock
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Hemodynamic Disorders, Thromboembolic Disease and Shock...
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PATHOLOGY
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Robbins and Cotran: Chapter 4 emodynamic Disorders, hromboembolic Diseases, and hock 1 Guia, Alexa M.
– 2D
Edema
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PATHOLOGY
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Robbins and Cotran: Chapter 4 emodynamic Disorders, hromboembolic Diseases, and hock 2 Guia, Alexa M.
– 2D
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Increased salt retention with obligate assoc. water o
Causes both increased hydrostatic pressure
PATHOLOGY
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Robbins and Cotran: Chapter 4 emodynamic Disorders, hromboembolic Diseases, and hock 3 Guia, Alexa M.
– 2D
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o
Due to vascular injury, including trauma, atherosclerosis, or inflammatory or neoplastic erosion of the vessel wall.
PATHOLOGY
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Robbins and Cotran: Chapter 4 emodynamic Disorders, hromboembolic Diseases, and hock 4 Guia, Alexa M.
– 2D
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o
Degrades adenosine diphosphate (ADP) o
PATHOLOGY
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Robbins and Cotran: Chapter 4 emodynamic Disorders, hromboembolic Diseases, and hock 5 Guia, Alexa M.
– 2D
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Concurrent activation of the coagulation cascade generates (stabilizes the platelet plug via two mechanisms) o
PATHOLOGY
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Robbins and Cotran: Chapter 4 emodynamic Disorders, hromboembolic Diseases, and hock 6 Guia, Alexa M.
– 2D
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o
Activated by binding to heparin-like molecules on endothelial cells (heparin – minimize thrombosis)
PATHOLOGY
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Robbins and Cotran: Chapter 4 emodynamic Disorders, hromboembolic Diseases, and hock 7 Guia, Alexa M.
– 2D
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o
Causes small vessel stasis
Deformed red cells in
o
PATHOLOGY
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Robbins and Cotran: Chapter 4 emodynamic Disorders, hromboembolic Diseases, and hock 8 Guia, Alexa M.
– 2D
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o
Can develop anywhere in the cardiovascular system o
Size and shape of thrombi depend on the site of origin and the cause o
PATHOLOGY
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Robbins and Cotran: Chapter 4 emodynamic Disorders, hromboembolic Diseases, and hock 9 Guia, Alexa M.
– 2D
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Altered t-PA production o
(systemic hypercoagulability) o
PATHOLOGY
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Robbins and Cotran: Chapter 4 emodynamic Disorders, hromboembolic Diseases, and hock 10 Guia, Alexa M.
– 2D
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Fat and associated cells released by marrow or adipose tissue injury may enter the circulation after the rupture of the marrow vascular sinusoids or venules
PATHOLOGY
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Robbins and Cotran: Chapter 4 emodynamic Disorders, hromboembolic Diseases, and hock 11 Guia, Alexa M.
– 2D
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Where tissue density limits the seepage of blood from adjoining capillary beds into the necrotic areas
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PATHOLOGY
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Robbins and Cotran: Chapter 4 emodynamic Disorders, hromboembolic Diseases, and hock 13 Guia, Alexa M.
– 2D
Endothelial injury
adult respiratory distress syndrome
Cause the failure of multiple organs (kidneys, liver, lungs, and heart)
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