Case study on CVA
January 21, 2017 | Author: molukas101 | Category: N/A
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A Case Presentation On Cerebrovascular Accident
Group J Marco Paul Velasco Precious Jane Parungao Rod Lambert de Leon Carla Aleja Abijay Mylene Narag Jenalin Quilang Krizzia Marie Palce Jessica Datul
OBJECTIVES
General Objective: At the end of the case presentation, the presenters together with the audience will enhance our understanding on the disease process of CVA, its nursing management and paves a way to us student-nurses appreciate our roles of being health care providers in the country’s quest for health progress and development.
Specific Objectives: • •
• •
• •
At the end of the presentation, presenters and audience will be able to: Define Cerebrovascular Accident. Discuss and interpret data gathered through theoretical analysis of Nursing History, Gordon’s 11 Functional Pattern, Physical Assessment and Laboratory Results. Explain the Anatomy and Physiology of Nervous System. Trace the Pathophysiology of Cerebrovascular Accdident. Create effective and efficient nursing care plan required by a patient with the above mentioned disease process. Discuss the medications taken by the client, its action, side effects and nursing responsibilities.
INTRODUCTION Cerebrovascular Accident Cerebrovascular Accident is a sudden loss of function resulting from disruption of the blood supply to a part of the brain. Stroke, also called brain attack or ischemic stroke, happens when the arteries leading to the brain are blocked or ruptured. When the brain does not receive the needed oxygen supply, the brain cells begin to die, a stroke can cause paralysis, inability to talk, inability to understand, and other conditions brought on by brain damage. Four types of stoke: 1. Cerebral Thrombosis- caused by blood clots. 2. Cerebral Embolism- caused by blood clots. 3. Cerebral Hemorrhage- caused by bleeding inside the brain. 4. Subarachnoid Hemorrhage- caused by bleeding inside the brain. Cerebral Thrombosis The most common type of brain attack. Occurs when a blood clot (thrombus) forms and blocks blood flow in an artery leading to the brain arteries primarily affected by atherosclerosis and more susceptible to blood clots. Most often occurs at night or in the morning when blood pressure in low. Often preceded by a transient ischemic attack (TIA) or “mini-stroke”. Cerebral Embolism Occurs when a wondering clot (embolus) or some other particle forms in a blood vessel away from the brain, usually in the heart. The clot then travels and lodges in an artery leading on the brain. Cerebral Hemorrhage Occurs when a defective artery in the brain busts. Subarachnoid Hemorrhage Occurs when a blood vessel on the surface of the brain ruptures and bleeds into the space between the brain and the skull. The World Health Organization (WHO) definition of stroke is “rapidly developing clinical signs of focal (or global) disturbance of cerebral function, with symptoms lasting 24 hours or longer or leading to death, with no apparent cause other than of (1) Noncommunicable disease. WHO Geneva (2) vascular origin” (3) By applying this definition transient ischemic attack (TIA), which is defined to less than 24 hours, and patients with stroke symptoms caused by subdural hemorrhage, tumors, poisoning, or trauma, are excluded. Based from the data gathered from TCGPH records section, there were 10 reported cases of CVA as of January 2009 until December 2009 comprises of 2 mortality cases and 8 morbidity cases.
Why this case? We have chosen this case as our topic during the case presentation because we would like that we, student-nurses, to be aware about CVA and also to broaden our knowledge about the management and treatment of this disease. Having awareness and gaining more knowledge about CVA would enhance our skills and attitudes in handling patients suffering from this disease.
This case serves as a challenge for us student-nurses to be committed and dedicated health professionals for the next days; we will take care of the health of the citizens.
PATIENT’S PROFILE
Name: Age: Gender:
I.M. 80 y/o Female
Civil Status:
Widower
Birth date:
Dec. 24, 1928
Nationality:
Filipino
Religion:
Roman Catholic
Address:
Ugac Norte, Tuguegarao City
Educational Background:
College Graduate
Occupation:
Retired Teacher
Date of admission:
November 19, 2009
Time of admission:
6:45 pm
Chief complaint:
loss of consciousness
Mode of arrival:
via stretcher
Admitting diagnosis:
HPN t/c CVA
Final Diagnosis:
CVA old recurrent Sepsis secondary to pneumonia NIDDM
Attending Physician:
Dr. Valeriano Combate, JR Dr. Marlene Cinco Dr. Gerardo Pagaddu, JR
Source of information: Hospital:
SO, patient’s chart, Record’s section TCGPH-Pay Ward
NURSING HISTORY Past Health History According to SO, when the patient suffered from headache, fever, and cough, patient takes over the counter drugs like paracetamol, biogesic, alaxan and solmux. Patient was diagnosed with Alzheimer’s disease on 2004, and undergone mastectomy when she was 42y/o. History of Present Illness According to SO, at the evening of November 19, 2009, 45 minutes PTC, SO noticed that patient was still sleeping at around 6:00pm. She then tried many times to wake up the patient and called her to eat but she did not receive any response. The SO was alarmed and decided to rush the patient to People’s Emergency Hospital and was admitted around 6:45pm. . At the age of 52 patient was hospitalized and diagnosed of HPN and manages it by taking maintenance drugs such as amlodipine, simvastatin & aspirin taken twice a day. Family Health History The patient has a history of Asthma on her paternal side. Her father died of Asthma and her mother died due to hypertension. Social Health History Patient is a retired teacher; she lives with her daughter and grand children. According to the SO before the patient was diagnosed of Alzheimer’s disease, the patient loves to mingle with her neighbors and loves to take care of her grand children. SO also verbalized that patient does not drink alcohol nor smoke cigarettes.
GORDON’S 11 FUNCTIONAL PATTERN Health Perception-Health Management Pattern Before Hospitalization During Hospitalization According to the SO, her mother According to the SO, she stated that her has been pampered starting when she mother is not in good condition. She believes was diagnosed with Alzheimer’s that doctors, nurses and other medical disease 5 years ago. When she members will help her mother to recover. SO suffered from the sickness, they also added that they obediently follow all the treated her immediately by taking OTC orders of the doctors. drugs for cough, colds and fever. With regards to her maintenance drugs to her hypertension, they give it at right time as prescribed. Nutritional- Metabolic Pattern Before Hospitalization According to the SO, her mother eats everything she wants and sees. She has no preference diet. She eats 3 times a day with mid afternoon snacks. She drinks 6-8 glasses of water a day. She has no difficulty in swallowing and has no allergy with any type of food. Elimination Pattern Before Hospitalization According to the SO, she defecates once a day with semi- formed and brown in color and being eliminated in morning. She voids 6-8 times a day with yellowish in color.
During Hospitalization Upon admission, the patient was inserted NGT and was ordered with PNSS 1liter to run for 8 hours. The diet was osteorized feeding with SAP.
During Hospitalization During our shift, the patient didn’t defecate. She has IFC connected to urine bag with 700 ml and yellow amber in color.
Activity Exercise Pattern Before Hospitalization
According to the SO, the patient is like a child. She plays with her neighborhood. Sometimes walking around their house. About her hygiene, they see to it that cleanliness must maintain to her.
During Hospitalization The patient is in comatose state. Student-nurses and SO initiated passive range of motion for her to exercise.
Sleep- Rest Pattern Before Hospitalization During Hospitalization According to the SO, her mother sleeps at Patient is comatose but can respond to around 8 in the evening and wakes up at physical stimuli. around 5 in the morning. She takes naps at afternoon. She has no rituals before sleeping she added. Cognitive Perceptual Pattern Before Hospitalization According to the SO, her mother is a retired teacher, she uses eyeglasses. She speaks dialects such as Ilocano, Tagalog and English.
During Hospitalization The patient responds to stimuli by means of rubbing her sternum for her to wake up.
Self- Perceptual Pattern Before Hospitalization The patient suffers from Alzheimer’s disease.
During Hospitalization The patient is comatose.
Role- Relationship Pattern Before Hospitalization According to the SO, before her mother was diagnosed with Alzheimer’s, she was a loving mother and responsible to her children. She provides their needs and sees to it that they are comfortable in their way of life.
During Hospitalization Due to her condition, her daughter stated that they will do all their best to take care of their mother. They will make sure to give back the care they have received from her.
Coping- Stress Pattern Before Hospitalization When her mother is tired, she sleeps for her to rest.
During Hospitalization During her present condition, she is in a stressful state. Her family is there to comfort and give her necessary needs just to show their love.
Sexual- Reproduction Pattern The patient has five children and had her menopause at the age of 50. Value Belief Pattern She is a Roman Catholic. When she was diagnosed with Alzheimer’s disease, her family never allowed her to go to mass, preventing her to lose her way home.
PHYSICAL ASSESSMENT Date Assessed: December 03, 2009, 5:15 PM Vital Signs: BP: 140/90 mmHg PR: 92 bpm RR: 23 cpm T: 36.8°C
• • • • •
•
General Appearance: ➢ Patient is lying on bed, comatose with ongoing IVF of PNSS 1L x 20 gtts/minute
at 500 cc level hooked at left metacarpal vein patent and infusing well. ➢ With NGT patent. ➢ With IFC connected to urine bag draining yellow amber. AREA ASSESSED
METHOD USED
NORMAL FINDINGS
Inspection
Fair complexion
ACTUAL
ANALYSIS
FINDINGS
SKIN –
Color
–
Texture
Inspection/ Palpation
–
Pale
Wrinkled Smooth
d/t decreased tissue perfusion and peripheral vasoconstriction
d/t loss of elastic fiber and decreased subcutaneous fat from hypodermis secondary to aging
Temperature d/t poor hygiene Inspection
–
Presence of rashes
Moisture
Palpation Normally warm
Cold and clammy
d/t peripheral vasoconstriction
d/t decreased
–
Turgor
Dry Palpation
Moist to dry
Sagged Palpation HAIR –
Snaps back to previous
activity of sebaceous and sweat glands secondary to aging
d/t loss of elastic fiber and decreased subcutaneous fat from hypodermis secondary to aging
distribution
Normal
– –
Texture Color
Inspection/
Evenly distributed
Evenly distributed
Palpation Normal NAILS –
–
–
Color of the nail bed Capillary refill time
Resilient Inspection
Inspection
Silky, resilient
Black w/ white hairs
Black
Shape d/t poor arterial circulation
EYES/EYEBROWS –
Shape
–
Symmetry
–
–
d/t decreased melanocyte production secondary to aging
Pallor
Inspection
Movement
Ability to blink
Pink transparent
d/t poor arterial circulation Delayed 4 sec.
Normal
Convex
Normal
Palpation Delayed 1-2 sec.
Palpation Normal CONJUNCTIVA –
Color
Convex
Round
Inspection
Normal Equal in size
Inspection
Round
PUPILS –
PERRLA
Inspection
Inspection –
Size of the pupil
EXTERNAL AUDITORY CANAL –
Equal in size
Symmetrical in movement
Symmetrical in movement
d/t decrease activity of CN V
Absence of blink d/t poor arterial circulation
Blinks involuntarily & bilaterally Pale Inspection
d/t compression of CN III
Hearing Pink-red
NOSE –
Symmetry
–
Color
Very slow to react to light
Inspection
Inspection
Response to penlight (dilates and constricts)
2mm
Normal
LIPS & MOUTH –
Symmetry
–
Color (lips)
–
Moisture
Normal Hears equally in both ears
Inspection
Hears equally in both ears Inspection Symmetrical
NECK
Inspection
Normal
Symmetrical
Same color as the face and neck
Normal
–
–
Symmetry Same color as the face and neck
Appearance Inspection
Symmetrical
Pale
THORAX –
Chest contour
Inspection
Symmetrical
d/t decrease oxygenation
d/t decreased salivary production r/t loss of vagal stimulation
Dry –
Clavicle
–
Chest wall
Normal
Pink Inspection
Normal –
Moist
Breathing pattern
Symmetrical Normal
ABDOMEN –
General contour
Palpation
Inspection
No distentions
Normal
Symmetrical Symmetrical
Normal
No distentions Inspection Prominent UPPER EXTREMITIES –
Inspection
Symmetry Inspection
–
Full chest expansion
Prominent
ROM
Normal Irregular
Inspection
LOWER EXTREMITIES –
Symmetrical
d/t decreased function of the medulla
Full chest expansion
Regular
Non-tender Normal
Size Inspection
–
Symmetry
Auscultation Percussion
–
ROM
Palpation
Non-tender
Normal
Symmetrical Inspection
Inspection/ Palpation
Symmetrical
(+) ROM upon movement
Normal (+) ROM upon movement
Normal Equal in size
Inspection
Inspection
Equal in size
Inspection
Symmetrical
Symmetrical
(+) ROM upon movement
(+) ROM upon movement
LABORATORY RESULTS HGT Date 11-21-09 6am
Normal
Result
Normal Range
284 mg/dl
80-120 mg/dl
Analysis
11-21-09 6pm
155 mg/dl
80-120 mg/dl
11-22-09 6am
186 mg/dl
80-120 mg/dl
11-22-09
153 mg/dl
80-120 mg/dl
11-23-09
170 mg/dl
80-120 mg/dl
11-24-09
215 mg/dl
80-120 mg/dl
11-27-09
172 mg/dl
80-120 mg/dl
11-28-09
152 mg/dl
80-120 mg/dl
11-30-09
120 mg/dl
80-120 mg/dl
12-01-09
133 mg/dl
80-120 mg/dl
Result
Normal Range
Na Date
Analysis
11-24-09
131 mmOl/L
135-145 mmOl/L
Normal
11-29-09
132 mmOl/L
135-145 mmOl/L
Normal
k Date
Result
Normal Range
11-24-09
3.0 mmOl/L
3.5-5.5 mmOl/L
11-29-09
4.0 mmOl/L
3.5-5.5 mmOl/L
Result
Normal Range
Analysis
Normal
CBC 11-20-09 Parameters
Analysis
WBC
12.4x103 /mm3
3.5-10
d/t increase pyrogens
RBC
3.83x106 /mm3
3.8-5.8
Normal
Hgb
11.4 g/dl
11.0-16.5
Normal
Hct
37.0%
35-50
Normal
PLT
188x103/mm3
150-390
Normal
INTAKE AND OUTPUT MONITORING SHEET 12-05-09 Intake
Output
Time
Oral
Parenter ral
Other Total s
Urine
Draina ge
Others
Total
7-3
500
100
600
600
600
3-11
1000
430
700
700
700
11-7
660
200
800
800
800
Total: 2890 Total: 2100 12-04-09 Intake
Output
Time
Oral
Parenter ral
Other Total s
Urine
Draina ge
Others
Total
7-3
720
100
75
895
200
250
3-11
1000
250
1250
500
500
11-7
600
250
850
200
200
Total: 2995 Total: 950 12-03-09 Intake
Output
Time
Oral
Parenter ral
Other Total s
Urine
Draina ge
Others
Total
7-3
750
350
75
1175
290
290
3-11
1000
200
4
1204
350
350
Total: 2379 Total: 640 12-02-09 Intake Time
Oral
Parenter ral
Output Other Total s
Urine
Draina ge
Others
Total
7-3
900
550
75
1525
790
790
3-11
832
120
75
1027
660
660
11-7
600
200
75
875
550
550
Total: 3427 Total: 2000 11-30-09 Intake
Output
Time
Oral
Parenter ral
Other Total s
Urine
Draina ge
Others
Total
7-3
600
340
940
1000
1000
3-11
890
475
1365
1100
1100
11-7
550
200
750
900
900
Total: 2055 Total: 3000 11-29-09 Intake Time
Oral
Parenter ral
3-11
800
300
Output Other Total s 1100
Urine
Draina ge
Others
400
Total 400
Total: 1100 Total: 400
11-28-09 Intake Time
Oral
Parenter ral
7-3
830
3-11 11-7
Output Other Total s
Urine
Draina ge
Others
Total
550
1380
1350
1350
1030
700
1730
600
600
700
700
1400
1650
1650
Total: 4510 Total: 3600 11-27-09 Intake Time
Oral
Parenter ral
7-3
1030
600
Output Other Total s
Urine
1630
1630
Draina ge
Others
Total 1630
3-11
600
450
1050
1050
1050
Total: 2680 Total: 2680 11-26-09 Intake
Output
Time
Oral
Parenter ral
Other Total s
Urine
Draina ge
Others
Total
7-3
860
475
1335
600
600
3-11
1250
400
1650
1250
1250
Total: 2985 Total: 1800 11-25-09 Intake
Output
Time
Oral
Parenter ral
Other Total s
Urine
Draina ge
Others
Total
7-3
770
350
1120
500
500
3-11
810
200
1010
800
800
11-7
800
200
1000
1250
1250
Total: 3130 Total: 2550 11-24-09 Intake
Output
Time
Oral
Parenter ral
Other Total s
Urine
Draina ge
Others
Total
7-3
715
400
1115
350
350
3-11
850
200
1050
1400
1400
Total: 2165 Total: 1750 11-23-09 Intake
Output
Time
Oral
Parenter ral
Other Total s
Urine
Draina ge
7-3
1030
200
1230
300
300
3-11
700
500
1200
600
600
11-7
600
750
1350
700
700
Total: 3780 Total: 1600
Others
Total
CRANIAL CT-SCAN Plain and contrast-enhanced axial tomographic sections of the head shows ill defined hypoattenvation in the both fronto-parietal periventrical and both occipital periventricular areas. The ventricles are unenlarged The midline structures are undisplaced The sulci and cisterns are prominent No abnormal extra-axial fluid collection detected The brain stem, pineal region and posterior fossa do not appear unusual The internal carotid basilar and vertebral arteries are calcified The sella turcica is not enlarged Soft tissue attenvation is noted in the right maxillary sinus IMPRESSION: Acute infarcts, both fronto-parietal periventricular and both occipital periventricular areas. Cerebral Atrophy Atherosclerotic Internal Carotid, basilar and vertebral arteries Sinusitis vs polyp, right maxillary sinus
ANATOMY AND PHYSIOLOGY Central Nervous System The Central Nervous System (CNS) is composed of the brain and spinal cord. The CNS is surrounded by bone-skull and vertebrae. Fluid and tissue also insulate the brain and spinal cord. Areas of the Brain The brain is composed of three parts: the cerebrum (seat of consciousness), the cerebellum, and the medulla oblongata (these latter two are “part of the unconscious brain”). The medulla oblongata is closest to the spinal cord and is involved with the regulation of heartbeat, breathing, vasoconstriction (blood pressure), and reflex centers for vomiting, coughing, sneezing, swallowing and hiccupping. The hypothalamus regulates homeostasis. It has regulatory areas for thirst, hunger, body temperature, water balance and blood pressure and links the nervous system to the Endocrine System. The midbrain and pons are also part of the unconscious brain. The thalamus serves as a central relay point for incoming nervous messages. The cerebellum is the second largest part of the brain, after the cerebrum. It functions for muscle coordination and maintains normal muscle tone and posture. The cerebellum coordinates balance. The conscious brain includes cerebral hemispheres, which are separated by the corpus callosum. In reptiles, birds, and mammals, the cerebrum coordinates sensory data and motor functions. The cerebrum governs intelligence and reasoning, learning and memory. While the cause of memory is not yet definitely known, studies on slugs indicate learning is accompanied by a synapse decrease. Within the cell, learning involves change in gene regulation and increased ability to secrete transmitters. The Brain During embryonic development, the brain first forms a tube, the anterior end which enlarges into three hollow swellings that form the brain, and the posterior of which develops into spinal cord. Some parts of the brain have changed little during vertebrate evolutionary history. Parts of the Brain as seen from the Middle of the Brain Vertebrate evolutionary trends include: 1. Increase in brain size relative to body size. 2. Subdivision and increasing specialization of the forebrain, midbrain and hindbrain. 3. Growth is relative in size of the fore brain, especially the cerebrum, which is associated with increasingly complex behavior in mammals. The Brain Stem and Midbrain The brain stem is the smallest and from an evolutionary viewpoint, the oldest and most primitive part of the brain. The brain stem is continuous with the spinal cord, and is composed of the parts of the hindbrain and midbrain. The medulla oblongata and pons control heart rate, constriction of blood vessels, digestion and respiration. The midbrain consists of connections between the hindbrain and forebrain. Mammals use this part of the brain only for eye reflexes.
The Cerebellum The cerebellum is the third part of the hindbrain, but it is not considered part of the brain stem. Functions of the cerebellum in clued fine motor coordination and body movement, posture and balance. This region of the brain is enlarged in birds and controls muscle action needed for flight. The Forebrain The forebrain consists of the diencephalon and cerebrum. The thalamus and hypothalamus are parts of the diencephalon. The thalamus acts as a switching center for nerve messages. The hypothalamus is a major homeostatic center having both nervous and endocrine functions. The Cerebrum The cerebrum, the largest part of the human brain, is divided into left and right hemispheres connected to each other by the corpus callosum. The hemispheres are covered by a thin layer of gray matter known as the cerebral cortex, amphibians and reptiles have only rudiments of this area. The cortex in each hemisphere of the cerebrum is between 1and 4mm thick. Folds divide the cortex into four lobes: occipital, temporal, pariental, and frontal. No region of the brain functions alone, although major functions of various parts of the lobes have been determined. The occipital lobe (back of the head) receives and processes visual information. The temporal lobe receives auditory signals, processing language and the meaning of words. The pariental lobe is associated with the sensory cortex and processes information about touch, taste, pressure, pain, and heat and cold. The frontal lobe conducts three functions: 1. Motor activity and integration of muscle activity 2. Speech 3. Thought processes Most people who have been studied have their language and speech areas on the left hemisphere of their brain. Language comprehension is found in Wernicke’s area. Speaking ability is in Broca’s area. Damage to Broca’s area causes speech impairment but not impairment of language comprehension. Lesions in Wernicke’s area impair ability to comprehend written and spoken words but not speech. The remaining parts of the cortex are associated with higher thought processes, planning, memory, personality and other human activities.
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